Instead of being thrown off the the trail by the differences, we should find the commonalities. Both models of aging- oxidative damage and telomere shortening cause chromosome disruption and bone marrow mesenchymal stem cell damage and depletion.
In the second of three interviews with Bill Andrews at the 2016 AMMG, we discuss the Henrietta Lacks (or HeLa) cervical cancer cells commonly used in cell research. Dr. Andrews also discusses why telomere erosion is less a factor in mice aging.
DISCLAIMER: This website is for educational purposes only and is not for advertising. Telomerase activators and nanovesicles are not FDA-approved to prevent or treat any disease and anecdotes are not scientific proof of efficacy. All patients were treated in the context of a fully informed and legally-protected patient physician relationship.
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Exosomes and TA-65 are not FDA-approved to prevent or treat any illness
