It would seem that it’s a “dog-eat-dog” work amongst dogs but when they are looking to be adopted and cared for by humans, they have co-opted the infant-parent cuteness that keeps us from abandoning our own young.
Google alerts for “telomeres” posted this interesting study validating my idea that replicative senescence is at the heart of liver disease. They engineered mice to have poor telomere repair and then watched as the large cell liver histology typical in hepatitis and cirrhosis developed.
Check out this very brief interview about how telomerase activation can enhance the anti-aging benefits of sleep:
When data from the Copehnhagen City Heart Study was published in 2013, they concluded that shorter telomeres in leukocytes was not associated with increased cancer after adjusting for age but that it WAS associated with reduced survival from cancer http://jnci.oxfordjournals.org/content/105/7/459.long
In 2015, the same data looked at certain gene tendencies that promote telomere shortening and concluded that cancer survival was IMPROVED when shortening was enhanced in the telomeres.
Contradiction? Not so fast.
I was contacted last week by a client who has gotten his diabetes under control at the age of 73. He couldn’t be happier but based on his past negative experiences and his desire to maintain respectability as a partner in a prominent law firm, he doesn’t want to endorse me or TA-65 openly.
DISCLAIMER: This website is for educational purposes only and is not for advertising. Telomerase activators and nanovesicles are not FDA-approved to prevent or treat any disease and anecdotes are not scientific proof of efficacy. All patients were treated in the context of a fully informed and legally-protected patient physician relationship.
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Exosomes and TA-65 are not FDA-approved to prevent or treat any illness
